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Neural Mechanisms of Cardiac Pain
Author Biography
Introduction
Anterolateral System
Somatic vs. Visceral Nociceptive Processing
Angina Pectoris
Sympathetic Sensory Innervation
Referred Pain
Vagal Sensory Innervation
Currently selected section: Other Ascending Pathways
Central Sensitization
Thalamus and Cerebral Cortex
Neurophysiology of Angina Pectorsis
Nausea and Vomiting

Dyspnea
Summary

 

Chapter 25:Neural Mechanisms of Cardiac Pain: Other Ascending Pathways
        

Other Ascending Pathways

Previously we have emphasized the probable role of the spinothalamic tract in transmitting noxious information from the heart. There are other ascending pathways that could also be involved, although none of them is as well-characterized as the spinothalamic tract.

The most prominent of these is the spinoreticular tract (Figure 1).

Spinoreticular neurons react similarly to spinothalamic neurons in response to noxious chemicals applied to the heart as well as to coronary artery occlusion (Blair et al., 1984 a & b). Considering the neuronal response patterns of the spinoreticular tract, as well as its projections, this pathway is likely involved in the transmission of neuronal activity that the brain ultimately interprets as angina pectoris.

Another possible pathway is the spinoparabrachial tract, which projects to the parabrachial region of the pons (Figure 9).

The cell bodies for the spinoparabrachial pathway are located primarily in the uppermost region of the dorsal horn (lamina I), and the axons terminate in the lateral parabrachial region (Blomquest et al., 1989; Cechetto et al., 1985; McMahon et al., 1985; Panneton and Burton, 1985; Menetrey and DePommery, 1991; Gauriau and Bernard, 2002). Neurons from the lateral parabrachial region project to the amygdala and the hypothalamus (ventromedial nucleus and retrochiasmatic region). These structures are likely to participate in the emotional-affective responses to noxious events, possibly including angina pectoris, as well as autonomic responses to noxious events.

Some additional pathways that could convey noxious information from the heart include the spinomesencephalic, spinosolitary, and spinohypothalamic tracts (Yezierski and Broton, 1991; Yezierski and Shwartz, 1986; Menetrey and Basbaum, 1987; Burstein, 1996), but very little information is known about their possible role in angina pectoris.

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